Document:Drug Consumption contents

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Contents

• Abstract
• 1. Virus-AIDS Hypothesis Fails to Predict Epidemiology and Pathology of AIDS
• 2. Definition of AIDS
  • 2.1. AIDS: 2 Epidemics, Sub-Epidemics and 25 Epidemic-Specific Diseases
    • 2.1.1. The Epidemics by Case Numbers, Gender and Age
    • 2.1.2. AIDS Diseases
    • 2.1.3. AIDS Risk Groups and Risk-group Specific AIDS Diseases
  • 2.2. The HIV-AIDS Hypothesis, or the Definition of AIDS
  • 2.3. Alternative Infectious Theories of AIDS
• 3. Discrepancies Between AIDS and Infectious Disease
  • 3.1. Criteria of Infectious and Noninfectious Disease
  • 3.2. AIDS Not Compatible with Infectious Disease
    • 3.3. No Proof for the Virus-AIDS Hypothesis
    • 3.3.1. Virus Hypothesis Fails to Meet Koch’s Postulates
    • 3.3.2. Anti-HIV Immunity Does Not Protect Against AIDS
    • 3.3.3. Antiviral Drugs Do Not Protect Against AIDS
    • 3.3.4. All AIDS-defining Diseases Occur in the Absence of HIV
  • 3.4. Noncorrelations Between HIV and AIDS
    • 3.4.1. Only about Half of American AIDS is Confirmed HIV-antibody-positive
    • 3.4.2. Antibody-positive, but Virus-negative AIDS
    • 3.4.3. HIV: Just One of Many Harmless Microbial Markers of Behavioral and Clinical AIDS Risks
    • 3.4.4. Annual AIDS Risks of Different HIV-infected Risk Groups, Including Babies, Homosexuals, Drug Addicts, Hemophiliacs and Africans, Differ over 100-fold
      • 3.4.4.1. Critically Ill Recipients of Transfusion
      • 3.4.4.2. HIV-infected Babies
      • 3.4.4.3. HIV-positive Homosexuals
      • 3.4.4.4. HIV-positive Intravenous Drug Users
      • 3.4.4.5. HIV-positive Hemophiliacs
      • 3.4.4.6. HIV-positive Teenagers
      • 3.4.4.7. HIV-positive General U.S. Population
      • 3.4.4.8. HIV-positive Africans
      • 3.4.4.9. HIV-positive Thais
    • 3.4.5. Specific AIDS Diseases Predetermined by Prior Health Risks
  • 3.5. Assumptions and Anecdotal Cases that Appear to Support the Virus-AIDS Hypothesis
    • 3.5.1. HIV is Presumed New Because AIDS is New
    • 3.5.2. HIV – Assumed to be Sexually Transmitted – Depends on Perinatal Transmission for Survival
    • 3.5.3. AIDS Assumed to be Proportional to HIV Infection
    • 3.5.4. AIDS Assumed to be Homosexually Transmitted in the U.S. and Europe
    • 3.5.5. AIDS Assumed to be Heterosexually Transmitted by African “Life-style”
    • 3.5.6. HIV Claimed to be Abundant in AIDS Cases
    • 3.5.7. HIV to Depend on Cofactors for AIDS
    • 3.5.8. All AIDS Diseases to Result from Immunodeficiency
    • 3.5.9. HIV to Induce AIDS via Autoimmunity and Apoptosis
    • 3.5.10. HIV Assumed to Kill T-cells
    • 3.5.11. Antibodies Assumed not to Neutralize HIV
    • 3.5.12. HIV Claimed to Cause AIDS in 50% Within 10 Years
    • 3.5.13. HIV Said to Derive Pathogenicity from Constant Mutation
    • 3.5.14. HIV Assumed to Cause AIDS with Genes Unique Among Retroviruses
    • 3.5.15. Simian Retroviruses to Prove that HIV Causes AIDS
    • 3.5.16. Anecdotal AIDS Cases from the General Population
  • 3.6. Consequences of the Virus-AIDS Hypothesis
• 4. The Drug-AIDS Hypothesis
  • 4.1. Chronological Coincidence Between the Drug and AIDS Epidemics
  • 4.2. Overlap Between Drug-Use and AIDS Statistics
  • 4.3. Drug Use in AIDS Risk Groups
    • 4.3.1. Intravenous Drug Users Generate a Third of All AIDS Patients
    • 4.3.2. Homosexual Users of Aphrodisiac Drugs Generate about 60% of AIDS Patients
    • 4.3.3. Asymptomatic AZT Users Generate an Unknown Percentage of AIDS Patients
  • 4.4. Drug Use Necessary for AIDS in HIV-Positives
    • 4.4.1. AIDS From Recreational Drugs
    • 4.4.2. AIDS from AZT and AZT Plus Confounding Recreational Drug Use
  • 4.5. Drug Use Sufficient for AIDS Indicator Diseases in the Absence of HIV
    • 4.5.1. Drugs Used for Sexual Activities Sufficient for AIDS Diseases
    • 4.5.2. Long-term Intravenous Drug Use Sufficient for AIDS-defining Diseases
  • 4.6. Toxic Effects of Drugs Used By AIDS Patients
    • 4.6.1. Toxicity of Recreational Drugs
    • 4.6.2. Toxicity of AZT
      • 4.6.2.1. AZT Not a Rational Anti-HIV Drug
      • 4.6.2.2. Toxicity of AZT in AIDS Patients and AIDS-free Persons
  • 4.7. Drug-AIDS Hypothesis Correctly Predicts the Epidemiology and Heterogeneous Pathology of AIDS
  • 4.8. Consequences of the Drug-AIDS Hypothesis: Risk-Specific Preventions and Therapies, but Resentment by the Virus-AIDS Establishment
• 5. Drugs and Other Noncontagious Risk Factors Resolve all Paradoxes of the Virus-AIDS Hypothesis
• 6. Why Did AIDS Science Go Wrong?
  • 6.1. The Legacy of the Successful Germ Theory: A Bias Against Noninfectious Pathogens
  • 6.2. Big Funding and Limited Expertise Paralyze AIDS Research

Tables and Figures

• Table 1. AIDS Statistics
• Table 2. Annual AIDS Risks of HIV-infected Groups
• Table 3. Immunosuppression in HIV-negative and -positive Hemophiliacs
• Figure 1. Determination of the Age of a Microbe in a Population Based on Farr’s Law

Notes, Acknowledgements, and References

• Note Added in Proof
• Acknowledgements
• References